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Commentary: Drinking beer may lead to higher levels of body fat than wine

Alcohol has long been considered one possible driving factor for obesity, but its link to higher levels of fat depends on the type of beverage consumed, says this researcher.

AMES, Iowa: Drinking beer and spirits are linked to elevated levels of visceral fat the harmful type of fat that is associated with an increased risk of cardiovascular disease, metabolic syndrome and other health complications – whereas drinking wine shows no such association with levels of this harmful fat and may even be protective against it, depending on the type of wine consumed. 

In fact, we found that drinking red wine is linked to having lower levels of visceral fat. These are some of the key takeaways of a new study that my colleagues and I recently published in the Obesity Science & Practice journal.

Although white wine consumption did not influence levels of visceral fat, our study did show that drinking white wine in moderation might offer its own unique health benefit for older adults: Denser bones.

We found higher bone mineral density among older adults who drank white wine in moderation in our study. And we did not find this same link between beer or red wine consumption and bone mineral density.

ALCOHOL A POSSIBLE FACTOR FOR OBESITY

Our study relied on a large-scale longitudinal database called the UK Biobank. We assessed 1,869 white adults ranging in age from 40 to 79 years who reported demographic, alcohol, dietary and lifestyle factors via a touchscreen questionnaire. 

Next, we collected height, weight and blood samples from each participant and obtained body composition information using a direct measure of body composition called dual-energy X-ray absorptiometry. Then, we used a statistical program to examine the relationships between the types of alcoholic beverages and body composition.

Ageing is often accompanied by an increase in problematic fat that can lead to heightened cardiovascular disease risk as well as a reduction in bone mineral density. This has important health implications given that nearly 75 per cent of adults in the United States are considered overweight or obese. 

Having higher levels of body fat has been consistently linked to an increased risk of acquiring many different diseases, including cardiovascular disease, certain types of cancer and a higher risk of death. And it’s worth noting that national medical care costs associated with treating obesity-related diseases total more than US$260.6 billion annually.

Considering these trends, it is vital for researchers like us to examine all the potential contributors to weight gain so that we can determine how to combat the problem. Alcohol has long been considered one possible driving factor for the obesity epidemic

Yet the public often hears conflicting information about the potential risks and benefits of alcohol. Therefore, we hope to help untangle some of these factors through our research.

(Photo: Pexels)

DIFFERENT ALCOHOLS HAVE DIFFERENT EFFECTS

There are many biological and environmental factors that contribute to being overweight or obese. Alcohol consumption may be one factor, although there are other studies that have not found clear links between weight gain and alcohol consumption.

One reason for the inconsistencies in the literature could stem from the fact that much of the previous research has traditionally treated alcohol as a single entity rather than separately measuring the effects of beer, cider, red wine, white wine, Champagne and spirits.

Yet, even when broken down in this way, the research yields mixed messages.

For example, one study has suggested that drinking more beer contributes to a higher waist-to-hip ratio, while another study concluded that, after one month of drinking moderate levels of beer, healthy adults did not experience any significant weight gain.

As a result, we aim to further tease out the unique risks and benefits that are associated with each alcohol type. Our next steps will be to examine how diet – including alcohol consumption – could influence diseases of the brain and cognition in older adults with mild cognitive impairment.

Brittany Larsen is a PhD Candidate in Neuroscience and Graduate Assistant at Iowa State University. This commentary first appeared in The Conversation.

Source: CNA/geh

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